German research found ways to prevent fatal blood transfusion complications German researchers announced that they have identified the pathogenesis of acute lung injury associated with blood transfusion and found a way to prevent such fatal blood transfusion complications. Andreas Greinach, a professor at the University of Greifswald in Germany, said in a press release that the research team he led discovered for the first time a blood group protein on human leukocytes that has not been known so far and was identified. Its molecular structure. During blood transfusion, if the donor's blood contains antibodies specific for HNS-3a, this antibody will often bind to this blood group protein of the recipient, causing leukocyte deformation and adhesion, blocking the microvessels of the lungs, resulting in acute lung injury . Professor Greinach said in a telephone interview with a reporter from Xinhua News Agency that, since the molecular structure of the aforementioned blood group protein was identified and related genes were found, the researchers artificially cultivated a large amount of this blood group protein with bacteria. A blood test based on this blood group protein can be used to find out which donors contain HNS-3a specific antibodies in their blood, thereby preventing acute lung injury associated with blood transfusion. He also said that this special blood test can be put into clinical use within a few months. Greinach also said that about one to two cases of acute lung injury occur in every 10,000 blood transfusions. About 5 out of every 100 people will produce HNS-3a specific antibodies in the body because of pregnancy or receiving blood transfusion. This antibody is not harmful to the carrier itself, but when he passes this antibody to others through blood donation Blood recipients will face threats. The acute lung injury associated with blood transfusion is mainly manifested by strong reactions such as respiratory distress, pulmonary edema, and hypoxia during or within 6 hours after blood transfusion.
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