Researchers at McMaster University in Canada found that there is a "central processor" inside the superbug that controls its pathogenic ability. The so-called "Central Processing Unit" is actually a small compound. Researchers have discovered that this compound is produced by Super Staphylococcus aureus in its drug-resistant form, which determines the strength and ability of the infection. Relevant experts believe that the discovery provides a new way to treat this deadly bacterial infection. Related articles were published in the latest issue of Science.
The most common superbug is methicillin-resistant Staphylococcus aureus (MRSA). In a narrow sense, MRSA refers to Staphylococcus aureus that is resistant to methicillin (a semi-synthetic antibiotic); broadly, it refers to Staphylococcus aureus that is resistant to one or more antibiotics. The pathogen is deadly and infectious. Although it is considered to be of low prevalence, cases have appeared in many countries.
Super germs can cause various infections and can resist the most effective antibiotics and drugs, and many antibiotics cannot kill it. Super germs can live and propagate in the human nasal cavity. Under normal circumstances, it only appears in the skin and nasal cavity, and usually heals itself without antibiotic treatment. However, if it encounters a patient after surgery or has a weakened immune system, it may cause infection in the body and cause pneumonia.
Associate Professor Nathan Magavi who presided over the research said that when they found that these small compounds in the superbugs ceased to function, the superbugs would lose their function and become infectious.
Researchers at McMaster University used advanced chemical prospecting methods to identify the small compounds that these scientists called "bacterial small molecule central processors" within the bacteria, thereby discovering the molecular links related to their formation. Later, in order to analyze the compound's function, scientists shut down its synthesis. Turning off its synthesis showed that this deadly bacterium was subdued and lost its ability to attack red blood cells.
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